Treatment of spleen tuberculosis. Tuberculosis Spleen: Symptomatics, Diagnostic Events, Features of Treatment Miliary Tuberculosis Spleen

The spleen performs important functions: filters blood and adjusts blood flow, contributes to blood clotting processes. Therefore, it is necessary to carefully monitor its work, since there are many diseases. One of them is an infectious disease (spleen tuberculosis). The causative agent is a koche wand. Initially, it hits the lungs. Further applies with blood flows to other organs.

Causes of pathology

The main cause of the miliary tuberculosis of the spleen is mycobacterium. There are many factors provoking the disease: weakened immunity and infection by mycobacteriums of other organs.

The first group includes:

1. Bad housing conditions. Long stay in high humidity and on the draft is poorly reflected in general well-being.
2. Harmful habits reduce the overall resistance of the body.
3. Speed \u200b\u200bup the spread of mycobacteria may be insufficient nutrition or harmful products. For healthy immunity it is important to get a necessary amount of vitamins with food. Wash clean water all products before use. Dirty water and food tolerate many malicious microorganisms. Among them may be a koche wand.
4. Little time for rest leads to the depletion of the body. This creates a favorable environment for the development of diseases.
5. Some features of professional activity (work in the situation of increased irradiation, air pollution, poor ecology).
6. Various mechanical injuries lead to a spleen tuberculosis.
7. Rarely, the tuberculosis wand can penetrate through damage to the skin.

The second group can be attributed to:

1. Contact with patients with tuberculosis.
2. Some diseases can provoke the propagation of tuberculosis (typhus, syphilis, immunodeficiency virus, hepatitis, infection in the biliary ways).

Mycobacteria tuberculosis falls into the body mainly by air-droplet.

Symptoms of tuberculosis spleen

Bacteria settle on the mucous membrane of the upper respiratory tract. Good immunity is able to destroy malicious microorganisms. If the immune system does not cope, mycobacteria is beginning to multiply, moving along the blood vessels. Next, the tuberculosis of the spleen can occur in two forms: chronic and acute. Each of these variations has its own characteristic symptoms.

In the chronic form of the disease, observed:

• subfebrile temperature (long-term increase in body temperature in the range of 37.1 - 38.0);
• splenomegali spleen to 3.5 kg;
• the number of leukocytes and blood platelets is dramatically increasing;
• cases of bleeding from nose and gums are rapid;
• anemia develops;
• sometimes hemoglobin rises to 200%;
• lowering appetite;
• strong fatigue;
• reduced performance;
• pain in the left hypochondrium;
• rarely liver tuberculosis.

In acute form, the disease is less common and differs in the following symptoms:

• high temperature (40.0);
• sharp change of blood composition;
• fast body exhaustion.

Diagnosis of the disease

First of all, the patient examines the specialists and direct on the tests. Common blood test and urine can show the presence of inflammatory processes. The following procedures are appointed:

1. Radiograph identifies foreign processes.
2. Computer tomography or magnetorezonance tomography.
3. Tuberculosis samples - help determine the level of immunity.
4. Ultrasound examination of the abdominal cavity shows the presence of changes in the spleen.
5. Fence and study of biological material for the presence of mycobacteria.
6. In rare cases, an electrocardiogram is prescribed.
7. Examination with the help of the camera.

A complex of diagnostic measures is selected individually to each patient.

How to treat tuberculosis spleen

With an isolated treatment of the disease, experts resort to splenectomy. This is an operation to remove the patient (in whole or in part). Before the operation of the patient is prepared:

1. Blood test for allergic reactions.
2. An electrocardiogram is removed, conduct an ultrasound study and a computed tomography of the abdominal cavity.
3. General analysis of blood and urine.
4. If necessary, vaccination is carried out.

It can be held in two ways: open operation and laparoscopy.

During the open operation, the surgeon makes an incision on the stomach. Gently breeds all the fabrics and removes the spleen. Further, the wound is well cleaned, stitched with threads and connect the brackets. Operating bandage is superimposed on the body.

Laparoscopy is carried out with a laparoscope. The doctor makes several incisions in which special tools are inserted. Then binds and cuts off the vessels and removes the spleen. The last step is sewn all cuts.

Medicia treatment

After identifying the reasons, tuberculosis treatment is prescribed. First of all, the patient is placed in the hospital. Specialists mainly apply a conservative method. It includes a complex of various drugs.

Prescribed antibiotic therapy from the first days. The preparation from tuberculosis - streptomycin is widespread. It helps to normalize all the indicators (reduce the spleen and improve the composition of the blood). The duration of taking antibiotics depends on the severity of the disease. The minimum course of treatment is 6 weeks.

Along with antibiotics immediately prescribe immunomodulators. Improving immunity contributes to rapid recovery, as it restores the protective functions of the body.

In support of medicines, the Vitamin complex is appointed. They fill the lack of important elements.

Full nutrition, one of the important stages. The exhaustion of the body leads to the rapid development of the disease.

Treatment of tuberculosis spleen folk methods

Nature has created a natural means to fight tuberculosis:

1. Cucumber juice mix with honey and take 3 tablespoons 3 times a day.
2. Oats pour milk, add sled. Put on a slow fire. As soon as the saber is dissolved, the mixture is removed and cool. Take 100 ml. 3 times a day.
3. 1 tablespoon of plantain pour 300 ml of boiling water, bitten and laughed 2 hours. Take 1 tablespoon 3 times a day before meals.
4. 20 gr. Aid to pour 300 ml of steep boiling water, put on a water bath for 20 minutes. Then bite and give cool. Take a decoction 3 times a day.

Folk methods are a good addition to the main treatment. Alone to deliver the spleen from tuberculosis they are not able.

Features of the treatment of spleen tuberculosis during pregnancy

With the presence of a spleen tuberculosis there are all chances to make and give birth to a healthy child. After identifying the disease, the doctor prescribes a complex of drugs. There is a certain group of medicines that can be used during pregnancy. After the birth of the child, the woman passes the test for the presence of mycobacteria. If such are not discovered, then experts are allowed to breastfeed.

However, if the disease occurs in combination with diabetes, doctors can advise to interrupt pregnancy.

Tuberculosis Splezenka - disease arising from the body infection with mycobacterium to tuberculosis. In the spleen, tuberculosis is very rare, more often the defeat occurs against the background of the existing process in the lungs.

Causes of the spleen tuberculosis:

• related immunity
• availability of infection in other organs

There are no specific symptoms of the spleen characteristic for tuberculosis, temperatures may occasionally. In addition, there is an increase in the spleen.

You can diagnose the tuberculosis of the spleen using biopsy, a radiographic study can also be indicative.

How to treat tuberculosis spleen?

Conducted according to the general principles of therapy of this disease, anti-tuberculosis drugs are prescribed. Throughout treatment, observation is recommended in dynamics using regular radiological studies. In addition, the reception of vitamins and immunostimulating drugs is required.

The surgical method of treatment is used for isolated spleen tuberculosis, after surgery, therapy is also shown by anti-tuberculosis drugs.

What diseases may be connected

Treatment of spleen tuberculosis at home

Treatment of tuberculosis spleen At home, it is possible after the expiration of the inpatient treatment of the underlying disease. Treatment is to continue receiving prescribed drugs.

What drugs to treat the tuberculosis of the spleen?

• Tubazid
• Salyzide
• and etc.

Treatment of tuberculosis spleen folk methods

Recipes of traditional medicine for the treatment of tuberculosis spleen does not exist. But since, this disease is secondary, the use of recipes intended for the treatment of lung tuberculosis is allowed.

• Mix 1 cup of honey and 300 grams of crushed aloe leaves, add 50 ml of water and put on fire. Cook on low heat for 2.5 hours, then cool. Take one tablespoon once a day
• Be sure to add garlic to the diet, eat 8-10 teeth during the day.
• Flowers of the richness (1 tbsp) Pour the glass of boiling water and insist one hour. Drink on a tablespoon 3 times a day.

Treatment of spleen tuberculosis during pregnancy

Treatment of tuberculosis spleen During pregnancy, it is carried out under the strict control of the phthisiatra. In modern pharmacology, there are drugs that do not have a significant teratogenic action on the fruit. Preparations are prescribed individually, after a thorough assessment of all risks for the child and the benefits for the mother.

What doctors to contact, if you have a spleen tuberculosis

Treatment of other diseases per letter - t

Treatment of thyrotoxicosis

Treatment of toxicodermia

Treatment of toxoplasmosis

Treatment of tracheitis

Treatment of trematodosis

Treatment of trichinelosis

Treatment of trichomoniasis

Treatment of thrombocytopenia

Clomes of the veins of the lower limb

Thrombus- blood clutch, is associated with intimate, dry, dull, compacted has a rough uneven surface, is easily deformed .

Causes of the formation of thromboms (Virchov Triad)

1. Damage to endothelium or endocardium. (Drystrophy damage, vessel inflammation)

2. Slow down blood flow. Meetings with a stitch of blood, in aneurysm, with angiosphasis in the area of \u200b\u200batherosclerotic plaque.

3. Blood coagulation disorders.

4 Stages of Education Thromba:

1. Adhesive to the wall, gluing platelets.

2. Coagulation of fibrinogen with the formation of fibrin.

3. Agglutination of red blood cells.

4. The settlement of plasma proteins.

In color 4 types:

White thrombusit consists of platelets, fibrin and leukocytes, formed in arteries and heart chambers. In the arteries arose. In atherosclerosis, in the aneurysms and in the chambers - with the heart of the midst., damage to valves during rheumatism or sepsis and the heart of Inevrisms.

Red Thrombcost. From platelets, fibrin and the large number of red blood cells. It is formed in the veins. (1-vein of a small pelvis, 2-veins Nizhn.Kon.) In Viennes, when inflammatory is inflaped, in laying patients, with zilk. With common dehydration-eseicosis. The mixed view of the thromba- consists of a head, body and tail.

Hyalin thrombus- Do not contain fibrin, occurs in the vessels of the microcirculatory bed.

In relation to the clearance of blood.

1. Prieuchenic

2. Finding

3. Character swarms are not attached to the wall, meetings in the heart chambers.

Exodes: fine blood clots are dissolved; organization; recacking; vascularization; Petrification.

Pulmonary embolism

Allocate 3 forms:

1. Lightning form -here, the thrombus stops either in the pulmonary trunk or in the bifurcation of the pulmonary artery. Death occurs after 15-20 minutes. The cause of death: a pulmonary reflex, which consists in the fact that the spasm of coronary arteries, pulmonary arteries and spasm of bronchi occurs reflexively.

2.Trecking form - Here, the thrombus stops in large branches of the pulmonary artery. Losts day. Death occurs due to the right-hand deficiency or due to massive hemorrhages in the lungs, acute breaths.

Z.Medlennoecurate form - Thrombus stops in the small branches of the pulmonary artery. Morphologically manifests the lung infarction.

Liver abscess.

The infection is entered in 2 ways: 1) on the biliary ways, 2) hematogenous via a portable vein system with intestinal infections.

The form of the organ is preserved, mass and dimensions are not increased. Coloring dark brown. At the bottom of the organ there is a deepening of oval shape 5x8 cm, a depth of 4 cm, the inner surface of which is lined with a connective tissue. The connecting tissue is located along the border of the deepening and in close proximity to it.

Pathological changes could develop as a result of the infectious lesion of the liver, which could be primary (independent disease) and be a manifestation of another disease. Extractive purulent inflammation develops, in which the granulation tissue shaft is formed around the focus of the infection, the accuracy of the abscess and supplying tissue protection cells (leukocytes) in the infectious hearth. Granulation fabric over time is replaced by a rough fiber connecting tissue. Capsules and acute abscess proceeds to chronic form.

Exodus: 1) favorable:a) the elimination of infectious agents and the organization of the abscess cavity (replacement of granulation tissue); b) chronic course of illness;

c) thickening in pus, turning it into necrotic child and petrification;

2) unfavorable:a) generalization of inflammation; b) breakthrough of the contents of the abscess into the abdominal cavity with the formation of a failure or into the lungs; c) lymphogenic and hematogenous distribution - septicopemia

Brewer pneumonia. (KP)

Truck (share, pleuropneumonium) is the primary infectious-allergic inflammation of the lungs.

Etiology: Pneumomococci 1-2 types, less often Chlebseyella (Fredlender wand)

Pathogenesis:related to the reactions of the GNT (hypersensitivity of the immediate type) in respiratory departments of the lungs. There are 2 mechanisms for the development of KP:

1. Pneumococci fall into the upper respiratory tract and cause sensitization of the entire body. Under the action of solving factors (supercooling, injury, etc.), the aspiration of the pathogen in the alveoli is aspiration, it causes a hypergic reaction with the development of KP

2. The causative agent from the nasopharynx falls into the pulmonary parenchyma and the RES authorities where immune reactions occur, then in blood flow, bacteremia and re-entering the pneumococcus in the lungs with blood leads to immunocomplex damage to the microcirculatory vessels of the alveoli with a characteristic exudative reaction.

In the development of bruboral pneumonia undergoes 4 stages:

1. Stradia tide (The 1st day lasts), the affected share is increased in size, it is sealed, dark red, sharply full. Under a microscope In Alveoli, serous fluid, a large number of pathogens, a bowel epithelium, and a pronounced full-voltage intervalolar abordination.

2. Stradia of the Red Heat - The next 2-3 days lasts. In this stage, the share of lungs becomes hepatic density, it is heavy, sinking in water. On Plevra, the imposition of fibrin, on the section of a dirty red color. The surface is fine-grained.

Gisto:fibrin-hemorrhagicservsudate.

Z. Stadia Gray Comparement- The next 4-6 days lasts.

Macro: The proportion is airless, increased in size, heavy, dense, on the cut gray, the surface of the cut is fine-grained. On Plegre Fibrin. Gisto: Fibrin-leukocytarial exudate.

4. Stradia permission. It begins with 9-11 days of the disease, lasts in different ways. In this stage, fibrinous exudate under the action of enzymes, neutrophils, macrophages diluted. The exudate is then removed according to the lymphatic vessels, the alveolar epithelium is regenerated. Macroscopic Share returns to normal sizes, graininess disappears.

Outcomes:

1. Full recovery with epithelium regeneration.

2. Perhaps the onset of death from pulmonary or rightful failure.

Z. Development of complications:

1. Pulmonary complications

1.1 Cartigination is an exudate organization in the Alveol.

1.2 Ecosis of the pulmonary fabric

1.3 abscesses

1.4 Empire Pleural cavity

1.5.Gangrena lung.

2. Wake-up complications:

2.1 Lymphogenic complications: - Voperation l / y. Mediosphere.-MediaSenit.-Pericarditis.

2.2Gematogenic complications: -Minegit, -Endocarditis.

Miliary tuberculosis spleen.

Hematogenic tuberculosis affects any organs and tissues, forming primary granulomas.

A source: Fucking primary tuberculosis complex, checkout.

Three varieties of hematogenic tuberculosis are distinguished:

1. generalized hematogenous tuberculosis;

2. Hematogenous tuberculosis with predominant lung damage;

3. Hematogenous tuberculosis with advantageous extrapulmonary lesions.

The macroscopic on the section of the spleen is visible multiple, diffusely located, projected, grayish color, dense consistency of the foci ("tubercles").

The structure of the primary tuberculosis granuloma.

It is usually revealed under a microscope, but they are able to merge, then they are visible macroscopic, they are called projected or miliar.

Micro: In the center of caseometric necrosis, the giant pyrogovan-langhans cells are scattered around epithelioid cells. Outside the layer of lymphocytes.

Exodes Granulul:

Sclerosis

Encapsulation

Suppuration

13. Polycystic kidney. (atrophy of pressure)

Polycystic kidney iscongenital bilateral disease. The basis is the absence of a message of some direct and convinced tubules. As a result, no passage of fluid, multiple cysts are formed in the kidneys. These cysts reduce normal nephrons. Nephrony are atrophy. In kidneys grows fat and connecting tissue. Macroscopic The kidneys are increased in size, bugs, have the form of grape clusters. On the section consist of a variety of cyst filled with translucent liquid.

Complications:

1. Urolithiasis.

2. Cancer tumors may develop.

3. Chronic renal failure.

4. Accession of infection and the development of pyelonephritis

Atrophy - a prominent decrease in the body at the expense of Parenhim.

May be:

Local

Total atrophy, or exhaustion, are found in the form of alimentary exhaustion (when fasting or disruption of food absorption); depletion with cancer cachexia (from Greek. Kakos bad, hexis condition); exhaustion at hypophized cachexia (Symon Disement with the lipophise damage); exhaustion under cerebral cachexia (lesion of the hypothalamus); depletion with other diseases (chronic infections, such as tuberculosis, brucellosis, chronic dysentery, etc.).

The appearance of patients in exhaustion is characteristic. There is a sharp altitude, the subcutaneous fatty fiber is absent; Where it saved, has a brownish color (accumulation of lipochroma pigment). Muscles are atrophichna, dry skin, shaky. Internal organs are reduced in size. In the liver and myocardium there are phenomena of brown atrophy (the accumulation of lipofuscin pigment in cells). In endocrine glands there are atrophic and dystrophic changes, expressed in a specifically degree depending on the cause of depletion. Plots of dead nerve cells are found in the cerebral cortex. Osteoporosis develops.

Local atrophy arises from various reasons. The following types of it differ:

1. Dysfunctional;

2. caused by blood supply deficiency;

3. pressure;

4. Neurotic;

5. Under the influence of physical and chemical factors.

Dysfunctional atrophy (atrophy from inaction) develops as a result of a decrease in the function of the organ.

Atrophy caused by the insufficiency of blood supply is developing due to the narrowing of the arteries that feed this body.

Atrophy caused by pressure develops even in organs consisting of dense tissue. Atrophy from pressure occurs in the kidneys with the difficulty of urine outflow.

Neurotic atrophy is due to violations of the bodies of the organ with the nervous system, which is due to the destruction of nerve conductors.

Atrophy under the influence of physical and chemical factors is a frequent phenomenon. Under the action of radiation energy atrophy is especially expressed in the bone marrow, genitals.

Heart hypertrophy.

Similar information.

The tuberculosis of the liver is a rare disease, but because no less dangerous. In case of late treatment or its absence, cases of fatal outcome are possible. This disease may be a manifestation of miliary tuberculosis or its local form in the absence of extrahepatic manifestations. The main form of liver damage during tuberculosis is granuloma.

The development of the disease of the tuberculosis of the liver occurs against the background of the general weakening of the human body.

The causes of such attenuation can be:

• overwork;
• small sleep;
• frequent stressful situations;
• adverse weather conditions;
• antisanitarian lifestyle;
• drug use.

To infect tuberculosis of the liver can be both an adult and a child. This is due to the end of the tuberculous chopstick. The most vulnerable to such a disease people who lead an unhealthy lifestyle (alcohol and tobacocco), as well as patients with diabetes, cancer or HIV infection.

Treatment takes a long period (up to one year), and the outlook outlook is about 85%.

The tuberculosis of the liver at the initial stage has symptoms similar to the pulmonary:

• long cough, dry in the first stage, and then with sputum release;
• increased drowsiness and sweating;
• the overall feeling of weakness;
• increased body temperature;
• weight loss;
• pain in the chest area;
• hemoop.

The main symptoms of liver tuberculosis at subsequent stages are:

• an increase in the size of the liver expressed by a change in dimensions in the lower part of the sternum;
• violation of the functions of the liver;
• sometimes this disease is accompanied by a jaundice;
• the presence of sharp pain in the liver area.

Symptoms of tuberculous liver disease can take place some time, but it is not worth waiting for this. Only timely treatment will help fully restore health.

Depending on the features of the course of illness and clinical symptoms, there are such types of liver tuberculosis:

• miliary tuberculosis;
• tuberculous granulomatosis;
• focal tuberculosis;
• tuberculous cholangitis;
• tuberculous pilephlegit.

More often than other forms occurs tuberculous granulomatosis. It occurs at the pulmonary or extractive form of tuberculosis.

This type of disease is characterized by an abundance of granuloma with necrotic mass in the center, and around - epitheliodic and lymphoid cells. In such granulomas, bacillos are concentrated with resistance to the effects of acid media.

The consequence of the flow of the disease is fibrosis (the formation of connective tissue around the granuloma occurs).

With focal tuberculosis, multiple tuberculosis of the liver is formed, around which the fibrous capsule is located, and in its center there are foci of necrosis. There is also an increase in the size of the liver. The course of the disease is characterized by the presence of weakness and fatigue, the absence of appetite and weight loss, the growth of the size of the spleen and the liver.

Miliary tuberculosis is accompanied by the formation of tubercles at once in several organs, it can be both acute and chronic.

Its main symptoms include:

• increased temperature and weakness (this is due to the fact that pathogenic bacteria can be constantly transmitted to the bloodstream);
• the presence of chills and cough;
• difficulties with breathing.

Macropreach "Miliary tuberculosis of the lung" is characterized by a low level of fracture, there is a large number of small projections, having a grayish-yellow shade.

The Miliary Tuberculosis Lung MicroPreparation is characterized by the formation of a large amount of granuloma, having casomic necrosis in the center of interlimoolar partitions and peribroscial tissue. Also in the peripheral sections there are cells of the Pirogov-Lanhhanza large size and an elevated level of lymphocytes.

Sometimes there is a disseminated tuberculous lesion of human organs, leading to the development of hepatic insufficiency. In this case, the impact of anti-tuberculosis drugs does not give results.

When casomic material gets into bile ducts, tuberculous cholangitis occurs. These ducts are destroyed against the background of the fact that the infection progresses, granulomas are formed in the bustling bubble and liver tissues.

The main sign of this disease is fever and lowered appetite (the body weight decreases), also possible the development of jaundice, and the rate of sedimentation of erythrocytes in the blood increases to 95 mm / h.

In the event of lesion of lymph nodes, caseaseous masses are capable of passing into portal veins, which leads to the occurrence of tuberculous pylephlebit. This course of the disease most often leads to a fatal outcome.

As with any other form of tuberculosis, the main aspect of the speedy recovery is the timely examination and diagnosis of the disease.

There are a number of techniques that allow you to reveal in the treatment of tuberculosis bad liver tests:

• computed tomography (CT);
• liver biopsy sample;
• magnetic resonance tomography (MRI);
• scanning the abdominal and chest cavity (ultrasound and photo).

The blood test with such a disease is ineffective, since it often gives an incorrect result. Sometimes the diagnosis of cholangiocarcinoma is also mistaken.

Listed methods make it possible to observe all changes in the structure of the liver and the neoplasm on its surface. According to the results of the procedure, an assessment of the disease is estimated, and the best way to confirm the detected disease is a thin game biopsy.

Female cases for the disease of the tuberculosis of the liver most often arise in people whose age is less than 22 years, which are taking treatment with steroids, sick HIV or AIDS, liver cirrhosis, or have liver failure.

It is necessary to treat the patient at the initial stages of the disease, then it will be as efficient as possible. The course of treatment takes a long time, and drugs carry a range of side effects. Most often, in the treatment of liver tuberculosis, doctors are written out: rifampicin, isoniazide, etcutol and pyrazinamide.

Another condition of high-quality treatment is the observance of the correct diet (diet No. 5 or 5a is assigned). Egg yolks, meat and fish with an abundance of fat, coffee, smoked and acute food should be excluded from the diet. To improve the exchange of amino acids in the body in the diet should be included: cottage cheese, liver, turkey, chicken, mushrooms, solid cheese, peas.

Proper nutrition in this form of tuberculosis is of great importance. It is the liver that passes through itself all the harmful components, making them harmless. Tuberculosis disrupts its functions, which can lead to poisoning.

The prevention of tuberculosis includes a regular passage of professional inspections and screening programs (allow you to identify the disease already at an early stage), children's vaccine vaccination or BCG-M is also carried out.

The lesion of the liver tuberculosis stick is sometimes complicated by other types of tuberculosis: spleen and bone marrow.

The main signs of such a combination are:

• changing the size of the spleen;
• wave fever;
• development of leukemoid reactions;
• painful sensations and severity in the left hypochondrium.

The risk of secondary liver fibrosis increases with prolonged leakage of the spleen tuberculosis. With such a course of the disease, it is subject to deformation, its surface acquires greater density and irregularity, a fighting process with surrounding tissues is possible. There is also a possibility that pneumonia will progress.

In the tuberculosis of the intestine can also meet the liver fatty dystrophy. As a result of such a disease, the liver increases in size, its density increases, dispersal disorders are observed.

The course of liver amyloidosis disease occurs without any features, this lies the reason for the difficulty of identifying it. The development of the disease occurs on the background of long-term bone tuberculosis or tuberculosis of light fibrous-cavernous form.

Viral hepatitis B and C and tuberculosis carry a special danger. Recently, there has been a tendency to an increase in the chronic form of hepatitis in combination with tuberculosis. This is due to the complication of socio-economic conditions, the growth of alcohol and drug dependence.

According to the results of the conducted studies, the dependence of the growth of diseases hepatitis B and C in combination with tuberculosis from the time of year was revealed. Most often an increase in the incidence rate is observed in the autumn and spring period.

Analysis of the basic characteristics of the combination of the auralar tuberculosis and hepatitis showed that the growth of morbidity is due to an increase in the patient's contingent, which previously suffered acute forms of hepatitis B and C, as well as an increase in the potential of infectious tuberculosis, including latent forms.

One of the side effects of the treatment of tuberculosis diseases is the likelihood of "medicinal disease" (toxic drug affects the body, as a result of which biochemical and functional structural changes occur in the body). Toxic hepatitis after anti-tuberculosis therapy occurs as a reaction to treatment.

Liver pathology arise in patients with tuberculosis in 15-20% of cases.

They are due to a number of factors:

• the effects of medicinal anti-tuberculosis drugs;
• chronic alcoholism;
• the presence of concomitant diseases (for example, hepatitis);
• drug addiction.

Hepatopathy is one of the most developed types of pathology in the Russian Federation. Different groups of patients have different frequencies and causes of violation of liver functions, and one of these groups are patients with tuberculosis patients.

Cirrhosis of the liver is able to develop due to prolonged effects of drugs in the treatment of tuberculosis. They are able to lead to the emergence of acute or chronic hepatitis. The cessation of drug reception leads to stabilization or regression of liver damage.

The use of hepatoprotectors in an anti-tuberculosis treatment complex contributes to the fact that the pathogenetic therapy will be more efficient. Such preparations (reamberin, remaksol, cytooflavin) allow the membrane of cells, which was destroyed during the treatment of tuberculosis.

Treatment of liver tuberculosis at home

After detection of the disease, the phthisiart determines the place where the patient will be treated:

• home treatment;
• hospital treatment.

The second method is preferable, since the treatment is more efficient, but the first option is also possible. Primary attention in the treatment is paid to the main foci of mycobacteria.

The choice of treatment strategy determines its effectiveness. This takes into account endogenous and exogenous factors that influence the distribution in the hepatic tissue of mycobacteria.

Tuberculosis is a fatal disease, therefore the use of traditional medicine is not enough for its cure. Mycobacteria affect the organism with a high degree of aggression, herbs extracts are incapable of sufficiently affect them. If the result and will be achieved, then briefly, and the consequences may be heavy.

In the complex with treatment under the supervision of the phthisiart, it is possible to apply:

• birch kidneys and sea buckthorn;
• marala root or saffelorovoid levses;
• rosehip;
• chinese lemongrass;
• blueberries;
• elecampane;
• hawthorn.

Based on the form of the disease, a mixture of aloe leaves and honey is used. With usual form, its use begins with the second month of treatment, with drug-sustainable - from the fourth.

Treatment of liver tuberculosis during pregnancy

Treatment of this disease during pregnancy occurs under the supervision of profile specialists. Of all the available drugs, the phthisiare appoints the safest, and the treatment process itself occurs according to the general rules.

A group of increased risk of exacerbation of the course of the disease during pregnancy is:

• pregnant women, who was less than a year ago, a tuberculosis was operating;
• pregnant under 20 and over 35 years old, which are already infected;
• healthy pregnant women who are in contact with tuberculosis patients.

The development of pregnancy is not a factor that contributes to the development of tuberculosis. However, clinical studies have shown that there is a small risk of exacerbation in the postpartum period.

Specialists with this form of the disease are a gastroenterologist and a hepatologist. Diagnosis of the disease is carried out by laparoscopy or using the liver biopsy (it has a special meaning when there is no symptom).

Analyzes show a slight change in functional samples, but there is an increase in levels:

• alkaline phosphatase;
• fractions gamma-glutamlitranspendase;
• alpha-2-globulin fractions.

Timely appeal to the doctor makes it possible to reduce the treatment time and speed up recovery. Do not deal with self-treatment, so as not to aggravate the course of the disease.

Complete Tuberculosis Free Online Test

Time limit: 0

Navigation (job numbers only)

0 of 17 tasks ended

Information

You have already passed the test earlier. You can't run it again.

The test is loaded ...

You must login or register in order to start the test.

You must finish the following tests to start this:

results

Time is over

• Congratulations! The likelihood that you are clogged to zero more tuberculosis.

  But do not forget to follow your body and regularly undergo medical examinations and no disease is not terrible!
  We also recommend reading the article by.

• There is a reason to think.

  It is impossible to say with the accuracy that you cannot say to tuberculosis, but this probability is, if it is not, then with your health, it is clear something wrong. We recommend that you immediately pass a medical examination. We also recommend reading the article by.

• Urgently consult a specialist!

  The likelihood of what you are amazed is very high, but remotely diagnose is not possible. You follow to you immediately get to a qualified specialist and pass a medical examination! We also recommend you to familiarize yourself with the article by.

1. With the answer
2. With a marker

1. Task 1 of 17

   1 .

   Is your lifestyle connected with heavy physical exertion?

2. Task 2 of 17

   2 .

   How often do you pass the test for the detection of tuberculosis (eg manta)?

3. Task 3 of 17

   3 .

   Do you carefully observe personal hygiene (shower, hands before meals and after walking, etc.)?

4. Task 4 of 17

   4 .

   Do you care about your immunity?

5. Task 5 of 17

   5 .

   Do your relatives sick or family members of the Tuberculosis?

6. Task 6 of 17

   6 .

   Do you live or work in an unfavorable environment (gas, smoke, chemical emissions of enterprises)?

7. Task 7 of 17

   7 .

   How often are you indoors with damp or dyed conditions, mold?

8. Task 8 of 17

   8 .

   How old are you?

The development of liver tuberculosis is provoked by hematogenous dissemination ("separation") mycobacteria of this dangerous infectious disease from other systems and organs. As a rule, this disease arises against the background of a long flow of tuberculosis and according to some data of statistics, about 80-99% of cases accompanies the tuberculosis of the intestine or. Among the total patients of phthisiators, tuberculous lesion of the liver tissues is detected from each hundredth of them.

The cunning of this form of tuberculosis is that the disease is difficult to diagnose in the early stages. How to notice the infection of the liver tissue with mycobacteriums? What methods of diagnosis and treatment are used to combat this dangerous disease?

Causes and varieties of the disease

This form of the disease develops, as a rule, on the background of the pulmonary tuberculosis, when mycobacteriums with blood flow propagate from pulmonary tissue to the liver.

The contact of mycobacteria and casease masses together with blood flow into hepatic fabrics is the main cause of the liver tuberculosis.

This process usually provokes the development of such forms of the disease:

• focal tuberculosis of the liver;
• tuberculous granulomatosis;
• miliary liver tuberculosis.

In more rare cases, caseometric masses falling into bile ducts or blood vessels of the organ provoke development:

• tuberculosis - with mycobacteria in bile paths;
• tuberculosis pilephlebit - when infection in the portal vein.

Miliary liver tuberculosis symptoms

More often this form is caused by a long flow of tuberculous lesion of the lungs or total dissemination of mycobacteria during bowel tuberculosis. Often, this ailment is revealed after the patient's death.

With miliar tuberculosis, the patient may present the following manifestations of the disease:

• the jaggility of the skin and scool;
• (sometimes);
• common symptoms of illness: feverish condition, chills, sweating.

The emergence of such symptoms of liver damage is provoked by tuberculosis granulomas that appear in the walls of the ducts and lobby of the liver. In addition, the yellowness of the skin and mucous membranes is caused by compression (squeezing) of the hepatic duct affected by mycobacterium lymph nodes.

When evaluating biochemical indicators of blood, functional hepatic samples are usually not violated. In some cases, the patient develops disseminated tuberculous lesion, accompanied by damage to the tissues of lungs, spleen and liver. Such a course of illness is expressed in Splenomegaly and the development of ascites. At the terminal stages of the disease, the patient detected signs.

With miliar tuberculosis liver, the use of anti-tuberculosis means is often ineffective.

Symptoms of tuberculosis granulomatosis

It is this kind of liver tissue tuberculosis that is most common. It can be detected in patients and with pulmonary, and with extractive forms of illness.

When analyzing the tissue of the organ affected by tuberculous granulomatosis, multiple granulomas are revealed with areas of necrosis in the center. It is usually precisely in granulations and contains micro-acid-resistant acid. Over time, there are still immature young connective tissues around the granuloma, and because of this, the organ is amazed by fibrous changes.

Clinic tuberculosis granulomatosis is extremely scarce. Increased liver in size is not detected from all patients.

Symptoms of focal tuberculosis

This type of disease is manifested by the formation of multiple or solitary tuberculos in the liver tissues. They are surrounded by a fibrous capsule, and necrosis zones can be detected in the center of the lesion.

When palpation of the abdomen of the patient, the doctor determines the hepatomegaly, and in some patients, tumor-like protruding on the surface of the liver is tumped. If the focal tuberculosis is manifested by the formation of insulated tuberculos, the disease is capable of a long time without manifest itself.

Subsequently, patients have the following complaints:

• pronounced weakness and a sharp decrease in tolerance to loads;
• deterioration of appetite;
• cachexia;
• fever.

When toughing the abdomen, hepatomegaly and splenomegaly is detected.

Symptoms of tuberculosis cholangita

After the receipt of caseous particles in the clearance of the bile duct in the patient, intrahepatic cholestasis and duct fabric begin to collapse. The patient has the following symptoms:

• fever;
• deterioration of appetite;
• jaundice.

In blood tests, it is determined by an increase in EFE to 95 mm / h, an increase in the level of gamma-glutorattranspend of serum and alkaline phosphotase.

Symptoms of tuberculosis Pilyphlebita

This type of damage to the liver tissues is developing with the damage to the portal lymph nodes and the intake of infected masses in the lumen of the portal vein. In most cases, such a development leads to the death of the patient.

What ailments can be combined with liver tuberculosis

If the tuberculosis of the liver is combined with the tuberculosis of the spleen, the patient bother gravity and stupid pain in the left hypochondrium.

In some clinical cases, tuberculous lesion of liver tissues is combined with bone marrow tuberculosis and spleen. With such a serious course of illness, the following symptoms are present in patients:

• wave fever;
• splenomegaly, accompanied by weight, discomfort and pain in the left hypochondrium.

If the bone marrow tissues are also subjected to infection, the patient has very pronounced leukemoid reactions and cytopenic syndromes:

• bleeding;
• next to infections.

In the tuberculosis of the spleen, the body ceases to be forgiven, and its surface becomes uneven, deformed and dense. In some cases, patients arise:

• manifestations of occupyless (inflammation of the outer shell of the organ);
• spikes with surrounding fabrics.

Due to changes in the pressure in the spleen vein, it develops, which can cause the development of liver fibrosis.

It can be provoked by a long flow of fibrous-cavernous tuberculosis of lungs or bone tissues. The manifestations of this liver pathology do not have specific features, and the disease is accompanied by standard symptoms for this disease.

In case of severe intestinal tuberculosis and other organs, the liver tissue dystrophy is possible.

• In patients with this pathology, digestion disorders arise, and the liver tissue becomes dense.
• The body increases in size and along with this symptom during the tack of the abdomen, an increase in the size of the spleen can be noted.
• In blood, hypergammaglobulinemia and hypoalbuminemia are often revealed.

Diagnostics

The following studies are assigned to confirm the diagnosis of "tuberculosis of the liver":

• a thin game;
• echography;
• diagnostic laparoscopy;
• blood tests.

Hepatic samples usually change slightly. Sore, level of alkaline phosphatase, gamma-glutamaltranspendase and alpha-2-globulin fractions are often rising. Some patients detected signs of anemia. Increasing the level of leukocytes for this disease is not typical.

Treatment

Therapy directly liver tuberculosis is of a secondary value, since the treatment will first of all be sent to the fight against the primary focus of infection. Some patients with such a disease can undergo a course of therapy at home, but more preferable is hospitalization of patients to a specialized hospital.

Typically assigned such anti-tuberculosis tools:

• Isoniazid;
• Pyrazinamide;
• Streptomycin;
• Rifampicin;
• Etcutol, etc.

The diagram of their reception and dosing is determined by the doctor individually and depend on the localization of the primary foci of tuberculosis. Usually, medication therapy continues for 1 year. Due to the high toxicity of anti-tuberculosis drugs, the patient during treatment can be advised to consult profile specialists:

• cardiologist;
• nephrologist;
• ophthalmologist and others.

During pregnancy, liver tuberculosis is treated according to generally accepted protocols, but a woman is prescribed more harmless drugs. In addition to the phthisiart, the observation of the obstetrician-gynecologist continues.

Popular treatment of liver tuberculosis, like other forms of this dangerous infection, is not recommended by specialists. Vegetable extracts cannot withstand this aggressive infection and do not have sufficient bactericidal action. As a rule, the passion for the folk methods of treatment of tuberculosis leads to a deterioration in the state of the patient, since only a temporary and short-term result is achieved or illness is progressing even more.

In addition to the above-described anti-tuberculosis drugs, it is assigned to maintain the functioning of the organ:

• glucocorticosteroids;
• protea inhibitors.

All patients with tuberculous liver damage are recommended by diet No. 5a or No. 5. The daily diet of such diets implies restriction of consumption in food:

• refractory fats;
• fried, sharp, salted, canned dishes;
• extractive substances.

Alcohol intake must be completely excluded. And with a sharp violation of the functions of the organ, the drug of the Sirepar is injected.

Forecast

The cirrhosis of the liver in the history of the patient with tuberculosis of this body shares its flow and worsens the forecast.

With timely identification of the disease, the forecast is usually favorable. However, the fact that the tuberculosis of the liver can continue to proceed asymptomaticly, often unhaps the forecast of the disease. Mortality in this form of tuberculosis according to various data is 15-40% (in developed countries, the mortality rate is much lower).